By John M. Tew Jr. M.D. (auth.), Mario Zuccarello, Joseph F. Clark, Gail Pyne-Geithman, Norberto Andaluz, Jed A. Hartings, Opeolu M. Adeoye (eds.)
The publication comprises forty eight articles awarded on the eleventh foreign convention on Cerebral Vasospasm held in Cincinnati, Ohio, united states, in July 2011. This choice of papers represents a cross-section of the large growth that has been made in the direction of an intensive realizing and powerful therapy of neurovascular occasions following aneurysmal subarachnoid hemorrhage, together with cerebral vasospasm. it's of curiosity to clinicians who desire to observe state of the art wisdom to their administration of this devastating situation and to uncomplicated scientists wishing to extend their knowing of cerebrovascular and neural pathophysiology relating to subarachnoid hemorrhage.
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Additional resources for Cerebral Vasospasm: Neurovascular Events After Subarachnoid Hemorrhage
Org) and expressed as pooled risk ratio (RR) estimates. Radiographic vasospasm was defined as a focal or generalized narrowing of cerebral arterial caliber on cerebral digital subtraction angiography or increased cerebral blood flow velocities as measured by transcranial Doppler (TCD). If angiographic vasospasm was categorized into no/mild, moderate, and severe vasospasm in included studies, the number of patients with moderate-to-severe vasospasm was included. TCD vasospasm was defined as a flow velocity of at least 120 cm/s or peak flow velocity of more than 200 cm/s.
Ca Introduction Patients who suffer subarachnoid hemorrhage (SAH) experience higher rates of morbidity and mortality compared to those who have other types of strokes . These poor outcomes may be due in part to cerebral infarction secondary to vasospasm or systemic inflammation, although the mechanisms by which poor clinical outcome occur due to these complications is unclear [11, 12]. Global brain atrophy in these patients may play a role as previous studies have linked regional brain atrophy in SAH with poor clinical outcome .
Crowley RW, Medel R, Kassell NF, Dumont AS (2008) New insights into the causes and therapy of cerebral vasospasm following subarachnoid hemorrhage. Drug Discov Today 13:254–260 4. Crowley RW, Medel R, Dumont AS, Ilodigwe D, Kassell NF, Mayer SA, Ruefenacht D, Schmiedek P, Weidauer S, Pasqualin A, Macdonald RL (2011) Angiographic vasospasm is strongly correlated with cerebral infarction after subarachnoid hemorrhage. Stroke 42:919–923 Randomised Trial of Clazosentan 5. Dorhout Mees SM, Rinkel GJ, Feigin VL, Algra A, van den Bergh WM, Vermeulen M, van GJ (2007) Calcium antagonists for aneurysmal subarachnoid hemorrhage.